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Joined 1 year ago
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Cake day: June 12th, 2023

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  • Most posters are talking about what natural disasters they experience and less about preparedness, so I’m going to take the preparedness angle:

    1. We have a go bag with medical supplies, very basic survival equipment, and non-perishable food.
    2. We have enough non-perishable food at home for my wife and I for about 3 months
    3. We have enough water for a week, and lifestraws to use local water supplies after that.
    4. We have basic survival things like hand crank chargers/radios, solar batteries, thermal blankets, etc.
    5. In the case of man made disaster (nuclear war) we have iodine pills.

    My take on survival stuff is to be prepared but not be a prepper. Some folks take this way too far. I feel everyone who builds a bunker and has a years worth of food is going to have someone fall flat on their house and it won’t matter anyway. That being said, I want to have enough to comfortably survive a week-month, and then after that things would be so fucked that all bets are off anyway.



  • Lots of the sub comments addressed this well, but I see no reason this couldn’t be the case with Crohn’s as well—maybe even more so than Lupus given the gut brain axis and microbiome effects. Likely there are a lot of different undiscovered pathways and molecular variants involved.

    Random side comment (and this is completely anecdotal) but I’ve diagnosed more new onset Crohn’s and UC since COVID than I had in my entire career previous to COVID. It makes me think there may be a component of COVID that’s similar enough to GI mucosa to cause autoimmune effects afterwards. Just need to wait to see the studies and incidence down the line.



  • I may not be answering this right, but the classification of autoimmune diseases—it’s likely a lot more complex than rheumatology would have you think.

    Take Lupus for example. Yes, there are bio markers that tend to be positive in cases of Lupus. However, there are varying degrees of positivity and a massive realm of variant symptoms. Prior to use of molecular assays in medicine we grouped these “somewhat similar” presentations into a single disease entity (lupus), but in reality it likely represents a cluster of similar diseases that are slightly different ways of the body attacking itself. The same is likely true to many other “vague” autoimmune diseases. It’s also true for the crossover between neurology and psychiatry. For example, based off of modern imaging we can tell that schizophrenia has an obvious organic root (massive brain structural changes), and this means it would probably be best owned by neurology like other neurodegenerative disease. Despite this, it is still owned by psychiatry and viewed by many (including some professionals) as a “chemical imbalance.” Schizophrenia is no more a dopamine excess than Parkinson’s is a dopamine lack—and we very much treat Parkinson’s as a neurodegenerative disease, not a psychiatric one. This difference is obviously due in part as much to historical classification as it is to the health equity problems surrounding schizophrenia patients.